Another notion food trolley popularized in the book Why Stomach Acid

This is the second article of a four part series on acid reflux and GERD. Read t he first article on the underlying cause, t he third article on the main stream medical treatments, and the final article on the myths of trigger foods and GERD diet that works without drugs.
I wrote about my own struggles with GERD in my first article and how I was able to control symptoms by modulating dietary carbohydrates. Some years later, I learned that I was infected with H. pylori. I was treated for the infection but there was no change in my susceptibility to acid reflux following treatment. My symptoms, though completely under dietary control, gradually reemerge when I consume too many carbohydrates over a period of 3-5 days, particularly food trolley those that are difficult to digest and absorb. Of course, my experience may not be typical food trolley of others with acid reflux and H. pylori. So, let’s take a closer look at this connection.
H. Pylori is a type of bacteria that has adapted through evolution to life in the stomach of approximately two thirds food trolley of all humans, though eradication efforts are making progress against this bacteria at least in developed countries. To survive in the stomach, the bacteria swims through mucous food trolley that lines the stomach by using multiple flagella , rotating hair-like appendages that provide locomotion. When H. pylori bacteria reach the stomach wall, they attach and begin secreting an enzyme called urease which converts urea to ammonia. The ammonia reduces the stomach acidity where the H. pylori is attached allowing this bacteria to survive and multiply. The biggest risks for people infected with H. pylori are stomach ulcers , duodenal ulcers , and stomach (gastric) cancer.
One study found that GERD patients were more likely to harbor H. pylori than non-GERD patients.[1] However, they did not find the same correlation in people with GERD who also had IBS. This makes little sense to me and casts some doubt on the validity of the findings.
On the other hand, numerous other studies have found that people harboring H. pylori are actually less likely food trolley to have GERD, including esophagitis and Barrett’s esophagus , two serious conditions food trolley arising from severe and chronic GERD. I will break down the studies into: GERD patients who had H. pylori but were treated for the infection. GERD patients with H. pylori infection compared to the same population without.
Studies on GERD patients where H. pylori was eradicated with antibiotics: H. pylori eradication had no impact on esophageal acid exposure or LES (lower esophageal sphincter) pressure.[2] H. pylori eradication resulted in no consistent change in gastroesophageal food trolley acid reflux.[3] No differences were detected in acid reflux before food trolley and after H. pylori eradication.[4] Eradicating H. pylori increased (not decreased) esophageal acid exposure and in some cases, food trolley worsened reflux symptoms.[5]
Studies on GERD patients with H. pylori infection compared to the same population without: The rate of H. pylori infection is lower in GERD patients compared to the general population. The absence of H. pylori is associated with more severe GERD. The results indicate a protective role of H. pylori against GERD.[6] H. pylori infection is inversely associated (protective) with GERD and GERD symptoms.[7] The H. pylori infection rate was lower in individuals with acid reflux compared to controls.[8] H. pylori infection was inversely associated with the risk and severity of reflux esophagitis, suggesting the organism may have a protective role against GERD.[9] H. pylori infection is associated with a lower risk of GERD.[10] H. pylori-negative patients had more severe esophagitis food trolley and were more likely to have Barrett’s esophagus.[11] H. pylori infection was inversely associated (protective) with Barrett’s esophagus food trolley and GERD symptoms were not associated with H. pylori infection.[12]
Conclusion : H. pylori infection is less common in patients with GERD, esophagitis and Barrett s esophagus. H. pylori infection is associated food trolley with less severe GERD symptoms and may have a protective role in GERD.
If H. pylori was a causative agent in GERD, one would expect that any decrease in H. pylori infection food trolley rates would also reduce the incidence of GERD, but the opposite is actually the case. As the worldwide incidence of GERD is increasing, infection rates of H. pylori are decreasing.[13] And as hospitalization rates have decreased for gastric and duodenal ulcers and gastric cancer, presumably due to H. pylori infection, hospitalization food trolley rates due to GERD and esophageal cancer, have risen significantly.[14]
Based food trolley on my review of the published literature and my own observations lead me to believe food trolley that H. pylori is not a causative factor in GERD in most people, and some strains of H. pylori may actually be protective for GERD. I will address H pylori s role in non-acid reflux below.
Another notion food trolley popularized in the book Why Stomach Acid